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primary antibodies (all in 1 : 1000 dilution) against inos, stata6, p-stat6, irf4, arg1, c-myc, and actb  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc primary antibodies (all in 1 : 1000 dilution) against inos, stata6, p-stat6, irf4, arg1, c-myc, and actb
    Primary Antibodies (All In 1 : 1000 Dilution) Against Inos, Stata6, P Stat6, Irf4, Arg1, C Myc, And Actb, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/irf4+primary+antibodies/Stat6+Antibody/pmc09410837-70-34-35
    Average 90 stars, based on 1 article reviews
    primary antibodies (all in 1 : 1000 dilution) against inos, stata6, p-stat6, irf4, arg1, c-myc, and actb - by Bioz Stars, 2026-10
    90/100 stars

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    Related Articles

    Western Blot:

    Article Title: Statins induce skeletal muscle atrophy via GGPP depletion-dependent myostatin overexpression in skeletal muscle and brown adipose tissue.
    Article Snippet: Myopathy is the major adverse effect of statins.. However, the underlying mechanism of statininduced skeletal muscle atrophy, one of statin-induced myopathy, remains to be elucidated.. Myostatin is a negative regulator of skeletal muscle mass and functions.



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    irf4 primary ab  (Cell Signaling Technology Inc)
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    Figure 7. Regulation of <t>IRF4</t> in CD8+ T cells by NAC1. CD8+ T cells were isolated from WT or NAC1−/−mice and analyzed for expression of IRF4. (A) Protein expression by Western blot. For the day 0 sample, T cells were not activated. For the day 3 sample, T cells were activated and cultured for 3 days. (B) mRNA expression by Q-PCR. CD8+ T cells had been cultured for 3 days, and then RNA was extracted, and qPCR was performed (ns, p > 0.05). (C) CHIP-seq analysis. WT CD8+ T cells were isolated from mice and CHIP was performed with anti-NAC1 and anti-IgG Abs (Input control). The sequenced data was visualized by IGV.
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    Figure 7. Regulation of IRF4 in CD8+ T cells by NAC1. CD8+ T cells were isolated from WT or NAC1−/−mice and analyzed for expression of IRF4. (A) Protein expression by Western blot. For the day 0 sample, T cells were not activated. For the day 3 sample, T cells were activated and cultured for 3 days. (B) mRNA expression by Q-PCR. CD8+ T cells had been cultured for 3 days, and then RNA was extracted, and qPCR was performed (ns, p > 0.05). (C) CHIP-seq analysis. WT CD8+ T cells were isolated from mice and CHIP was performed with anti-NAC1 and anti-IgG Abs (Input control). The sequenced data was visualized by IGV.

    Journal: Viruses

    Article Title: Expression of NAC1 Restrains the Memory Formation of CD8 + T Cells during Viral Infection.

    doi: 10.3390/v14081713

    Figure Lengend Snippet: Figure 7. Regulation of IRF4 in CD8+ T cells by NAC1. CD8+ T cells were isolated from WT or NAC1−/−mice and analyzed for expression of IRF4. (A) Protein expression by Western blot. For the day 0 sample, T cells were not activated. For the day 3 sample, T cells were activated and cultured for 3 days. (B) mRNA expression by Q-PCR. CD8+ T cells had been cultured for 3 days, and then RNA was extracted, and qPCR was performed (ns, p > 0.05). (C) CHIP-seq analysis. WT CD8+ T cells were isolated from mice and CHIP was performed with anti-NAC1 and anti-IgG Abs (Input control). The sequenced data was visualized by IGV.

    Article Snippet: The IRF4 primary Ab used is rabbit anti-mouse IRF4 Ab (CST #62834T).

    Techniques: Isolation, Expressing, Western Blot, Cell Culture, ChIP-sequencing, Control